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Heart Failure

A pump that cannot keep up. Which side fails determines where the fluid backs up — and that one idea explains nearly every sign you will assess.

Physiological AdaptationReduction of Risk Potential

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A broken pump with fluid backing up into the lungs above and the legs below, while only a weak trickle leaves it forward.

Pink frothy sputum is flash pulmonary edema

Critical

Sit the patient upright with legs dependent, apply oxygen, and escalate immediately. This deteriorates within minutes.

Reference values

BNP — heart failure unlikely

< 100pg/mL

Points away from heart failure as the cause of this patient's breathlessness. Look for another explanation.

BNP — strongly suggestive

> 400pg/mL

Released when the ventricle is stretched, so it tracks volume overload and falls as congestion resolves.

Report weight gain

> 1 kg / day

Or 2.5 kg (5 lb) in one week

Hold digoxin if apical pulse

< 60bpm

Count a full minute at the apex before every dose. Hold and report rather than estimate from a radial pulse.

Normal ejection fraction

55 – 70%

The share of blood the left ventricle ejects each beat. Below 40% defines reduced-ejection-fraction failure.

01One principle, both sides

Heart failure is a pump problem, and fluid backs up behind the side that is failing. If you hold onto that single sentence you can reconstruct the entire symptom picture without memorising two separate lists.

The left ventricle sends blood to the body. When it fails, blood backs up behind it into the lungs. The right ventricle sends blood to the lungs. When it fails, blood backs up behind it into the body.

Plate HF-01

Direction of congestion

Left-sided failure

Backs up into — The lungs

LUNGSLVRVBODY
  • Crackles
  • Orthopnea
  • PND
  • Pink frothy sputum
  • S3 gallop

Right-sided failure

Backs up into — The body

LUNGSLVRVBODY
  • JVD
  • Peripheral edema
  • Hepatomegaly
  • Ascites
  • Weight gain
Fluid backs up behind the side that is failing. Left ventricle sends blood to the body, so its failure congests the lungs. Right ventricle sends blood to the lungs, so its failure congests the body.

02Left-sided failure — the lungs fill

Everything here is respiratory:

  • Dyspnea on exertion, progressing to dyspnea at rest
  • Orthopnea — breathless lying flat, so the patient sleeps on extra pillows
  • Paroxysmal nocturnal dyspnea — waking abruptly gasping a few hours into sleep
  • Bilateral crackles that do not clear with coughing
  • A persistent dry cough, and in severe decompensation pink frothy sputum
  • S3 gallop
  • Restlessness and confusion as cerebral perfusion drops

Pink frothy sputum means alveoli are filling with fluid. That is flash pulmonary edema and it is an emergency.

03Right-sided failure — the body swells

Everything here is systemic congestion:

  • Jugular venous distension
  • Dependent peripheral edema — ankles in a walking patient, sacrum in a bedbound one
  • Hepatomegaly, ascites, and right upper quadrant fullness
  • Weight gain, which appears before visible swelling
  • Nausea and poor appetite from gut congestion

The most common cause of right-sided failure is left-sided failure. Over time the backed-up pressure in the lungs wears out the right ventricle too.

04Why daily weight beats everything else

Edema is a late sign. A patient can retain litres of fluid before you can see or press a mark into their ankle. The scale catches it days earlier.

One kilogram of weight gain equals roughly one litre of retained fluid. Weight is taken at the same time each morning, after voiding, in similar clothing, on the same scale.

Teach the patient the reporting threshold explicitly: more than 1 kg (2–3 lb) in a day, or 2.5 kg (5 lb) in a week, gets a phone call. That single habit prevents a large share of readmissions.

05Medications and what to watch

ACE inhibitors (-pril) reduce afterload. Watch for a persistent dry cough, hyperkalemia, and first-dose hypotension. A cough that will not settle is a common reason for switching to an ARB (-sartan).

Beta blockers (-olol) reduce workload long term but can worsen symptoms initially. Hold and reassess for bradycardia.

Loop diuretics such as furosemide pull fluid off quickly. Watch for hypokalemia, hypotension, and ototoxicity when given rapidly IV.

Digoxin strengthens contraction and slows rate. Take an apical pulse for a full minute and hold for a rate under 60. Early toxicity is easy to miss because it looks like ordinary malaise — nausea, anorexia, fatigue, and characteristically yellow-green visual halos.

Digoxin toxicity hides behind hypokalemia

Watch

A low potassium increases myocardial sensitivity to digoxin. Toxicity can occur at a digoxin level within the therapeutic range.

Nursing actions

  1. 01Position upright — high Fowler's — to reduce venous return and ease work of breathing.
  2. 02Weigh daily at the same time, after voiding, before breakfast.
  3. 03Track strict intake and output; correlate with the weight trend, not against it.
  4. 04Auscultate lung fields and assess for JVD and dependent edema each shift.
  5. 05Teach sodium restriction and the specific weight threshold that triggers a call.
  6. 06Pace activity with planned rest periods to limit myocardial oxygen demand.

Sources

SourceLicense
Open RN — Nursing Fundamentals / Pharmacology (CC BY 4.0)Informed scope and sequence. Prose written originally for this portal.CC-BY-4.0
MedlinePlus / NIHPublic Domain (US Gov)