Nursing Nerds

Respiratory Failure, Shock and Critical Care

Book 08 · Page 66 / 15

Read the hemodynamics, not the history

Airway cross-sections comparing reversible asthma narrowing with the fixed obstruction of chronic obstructive pulmonary disease.
Fig 9.6 · pending clinical review

The shock matrix

THE TANK IS EMPTY HypovolemicTHE PUMP OR PATH IS BLOCKED Cardiogenic and obstructiveTHE PIPES ARE OPEN Distributive
Preload · neck veins↓ Flat↑ Distended↓ relative, flat
Afterload · systemic vascular resistance
Cardiac output↔ or ↑ early, ↓ late
Heart rate↑ (↓ in neurogenic)
SkinCool, pale, clammyCool, mottledWarm, dry or flushed early
Pulse pressureNarrowNarrowWide
What is actually wrongVolume has left the circuit: hemorrhage, burns, vomiting, diarrhea, third spacingCardiogenic is the muscle failing. Obstructive is tension pneumothorax, cardiac tamponade, or massive pulmonary embolismVasodilation and capillary leak: septic, anaphylactic, neurogenic
First moveReplace volume and control the lossRelieve the obstruction, or support the pumpFluid plus a vasoconstrictor, and source control
What makes it worsecrit Vasoconstrictors given instead of fluidcrit Aggressive fluid poured into a heart or a path that cannot move itcrit Fluid alone when the vessels are the problem

The three flavors of distributive shock

  • Septic. An infection source plus vasodilation, capillary leak, and a rising lactate. Skin warm and flushed early, cool and mottled once it has gone on too long. First move is fluid, cultures, antibiotics, and a vasopressor if the pressure will not hold.
  • Anaphylactic. Everything above plus stridor, hoarseness, urticaria, angioedema, and bronchospasm within minutes of an exposure. First move is intramuscular epinephrine into the anterolateral thigh, before oxygen, before access, before anything else. Then airway, oxygen, supine with legs elevated, fluid, and repeat dosing if there is no response.
  • Neurogenic. The exception that breaks the tachycardia rule. Loss of sympathetic outflow after a cervical or high thoracic injury gives hypotension with bradycardia and warm, dry skin below the level of injury. Fluid is given cautiously, a vasoconstrictor is usually required, and spinal precautions are maintained throughout. Injury classification is → BOOK 10 · P5.

The three-question read

Neck veins flat or full. Skin cool or warm. Rate fast or slow. Three answers name the family before a single word of history is taken.

Memory hook: FLAT · COOL · FAST is the hypovolemic signature. Change one variable and you change columns. Full neck veins move you to column 2. Warm skin moves you to column 3. Slow rate with warm skin moves you to neurogenic.

Margin notewarm and hypotensive is not reassuring.

Watch outA patient can be hypotensive and warm to the touch and still be dying faster than the cold one. Skin temperature is a clue to the mechanism, never a measure of how sick.

Cardiogenic shock is placed here by signature only. The failing heart, its rhythms, and its drugs are → BOOK 08 · P3. Vasopressor pharmacology and titration are named, never taught: → BOOK 05 · P6 and → BOOK 06 · P7. The definitional four-shock map is → BOOK 01 · P44.

TakeawayNeck veins and skin temperature sort every shock into one of three families before you know a single diagnosis.